Number of children and maternal mental health in the context of China’s fertility policy transition: the moderating effect of employment status and the mediating effect of family environment
Iron dyshomeostasis in neuropsychiatric disorders
Coping under pressure: police-specific stressors and mental health in Catalonia police forces
Is AI actually improving healthcare?
Nature Medicine, Published online: 21 April 2026; doi:10.1038/s41591-026-04329-2
Is AI actually improving healthcare?
The Q-MONSTAR consortium: advancing fault-tolerant quantum computing for precision oncology
Nature Medicine, Published online: 21 April 2026; doi:10.1038/s41591-026-04346-1
The Q-MONSTAR consortium: advancing fault-tolerant quantum computing for precision oncology
Exposure to negative physical and social factors accelerates brain aging
Nature Medicine, Published online: 21 April 2026; doi:10.1038/s41591-026-04348-z
In a study that included 18,701 people from 34 countries, 73 aggregated physical and social exposomes exhibited nonlinear, synergistic effects that accelerated brain aging. In some cases, these effects were comparable to or stronger than those of mild cognitive impairment and dementia. Environmental inequities might shape brain aging and demand multisectoral and structural policy responses.
Show us the evidence for the value of medical AI
Nature Medicine, Published online: 21 April 2026; doi:10.1038/s41591-026-04389-4
Claims that medical AI is improving care must be backed by appropriate evidence.
Modifying exposure to plastic-associated chemicals in daily living
Nature Medicine, Published online: 21 April 2026; doi:10.1038/s41591-026-04349-y
A study shows that extensively limiting plastic use in food systems, diet and daily life can reduce exposure to plastic-associated chemicals such as phthalates and bisphenols, indicating that large-scale regulatory actions are needed for true public health impact.
Biophysical modeling of excitation/inhibition balance and conversion to psychosis in the clinical high risk syndrome
Reduced mismatch negativity (MMN) and P300 event-related potential (ERP) components are widely replicated in schizophrenia and are also observed in individuals at clinical high risk for psychosis (CHR-P) who subsequently convert to psychosis. It is unknown whether they reflect changes in excitatory and/or inhibitory synaptic function, both implicated in schizophrenia and considered potential drug targets.

